Complete Information on Cold agglutinin disease

>autoimmune hemolytic anemia are caused by cool
Cold Agglutinin Disease, a rare blood disorder, is aagglutinins only. Symptoms of this disorder may
type of autoimmune hemolytic anemia. Cold agglutinininclude tiredness; anemia caused by reduced levels of
disease, also called cold antibody disease. Coldcirculating crimson blood cells; jaundice characterized
agglutinin disease (CAD) usually develops as a resultby relentless yellowing of the rind, mucous
of the production of a specific IgM antibody directedmembranes and whites of the eyes; and/or
against the I/i antigens (precursors of the ABH andperspiration and coolness of the fingers and/or toes
Lewis blood group substances) on RBCs.and spotty bluish or crimson discoloration of the rind
It is caused by the body mistakenly makingof the fingers toes, ankles, and wrists. Cold
antibodies (of the IgM type) against a component ofagglutinins are seen in CANOMAD syndrome.
red blood cells. Cold agglutinin disease is classified asCANOMAD syndrome is described by gait and
primary or secondary. Primary cold agglutinin diseaseupper-limb ataxia.
is usually associated with monoclonal cold-reactingCranial nerve involvement with external
autoantibodies.ophthalmoplegia. More than 80% of patients have
Cold agglutinin disease occurs in the pediatricevidence of hepatitis C infection. Interferon and
population but is more frequent in the elderlyinterferon plus ribavirin have been shown to produce
population.Primary cold agglutinin disease is chronic andserologic responses. Conventional medical treatments
occurs after the fifth decade of life, with a peakmay help relieve the symptoms of cold agglutinin
incidence at approximately age 70 years. Secondarydisease. When vasculitis is active, corticosteroids are
cold agglutinin disease may be associated with eitheroften required to permit healing of ulcers in the skin
monoclonal or polyclonal cold-reacting autoantibodies.or to treat the membranoproliferative
Secondary cold agglutinin disease is predominantlyglomerulonephritis that is seen, thereby preventing
caused by infection. Secondary cold agglutinin diseaseloss of renal function. Rituximab therapy has been
in children and young adults is usually transient and isfound to be effective in mixed cryoglobulinemia, with
caused by infection. Secondary cold agglutinin diseasedecreases in cryoglobulin values and improvement in
primarily occurs in children and young adults.complement values.
Overall, roughly 7-25 percentage of cases of